• 研究成果概要・研究業績
  • 文部科学省
HOME研究成果概要・研究業績 > 研究業績 > 平成24〜25年度: 公募研究04
研究業績

平成24〜25年度: 公募研究04

管形成過程における紡錘体配向の変換機構
研究代表者: 松本 邦弘

英文原著論文

  • Morioka, S., Broglie, P., Omori, E., Ikeda, Y., Takaesu, G., Matsumoto, K., Ninomiya-Tsuji, J. TAK1 kinase switches cell fate from apoptosis to necrosis following TNFα stimulation. J. Cell. Biol., (in press).
  • Hattori, A., Mizuno, T., Akamatsu, M., Hisamoto, N., Matsumoto, K. The C. elegans JNK signaling pathway activates expression of stress response genes by derepressing the Fos/HDAC repressor complex. PLoS Genet. 9: e1003315, 2013.
  • Uno, M., Honjoh, S., Matsuda, M., Hoshikawa, H., Kishimoto, S., Yamamoto, T., Ebisuya, M., Yamamoto, T., Matsumoto, K., Nishida, E. A fasting responsive pathway that extends life-span in C. elegans. Cell Reports 3: 79-91, 2013.
  • Inoue, A., Sawatari, E., Hisamoto, N., Kitazono, T., Teramoto, T., Fujiwara, M., Matsumoto, K., Ishihara, T. Forgetting in C. elegans is accelerated by neuronal communication via the TIR-1/JNK-1 pathway. Cell Reports 3: 808-819, 2013.
  • Li, C., Hisamoto, N., Nix, P., Kanao, S., Mizuno, T., Bastiani, M., and Matsumoto, K.* The growth factor SVH-1 regulates axon regeneration in C. elegans via the JNK MAPK cascade. Nature Neurosci. 15: 551-557, 2012.
  • Pastuhov, S. Iv., Fujiki, K., Nix, P., Kanao, S., Bastiani, M., Matsumoto, K.*, Hisamoto, N. Endocannabinoid-Goα signalling inhibits axon regeneration in Caenorhabditis elegans by antagonizing the Gqα-PKC-JNK cascade. Nature Commun. 3: e1136, 2012.
  • Morioka, S., Inagaki, M., Komatsu, K., Mishina, Y., Matsumoto, K., Ninomiya-Tsuji, J. TAK1 kinase signaling regulates embryonic angiogenesis by modulating endothelial cell survival and migration. Blood 120: 3846-3857, 2012.
  • Ota, S., Ishitani, S., Shimizu, N., Matsumoto, K., Itoh, M., Ishitani. T. NLK positively regulates Wnt/β-catenin signaling by phosphorylating LEF1 in neural progenitor cells. EMBO J. 31: 1904-1915, 2012.
  • Omori, E. Inagaki, M., Mishina, Y., Matsumoto, K., Ninomiya-Tsuji, J. Epithelial TAK1 kinase is activated through two independent mechanisms and regulates reactive oxygen species. Proc. Natl. Acad. Sci. USA. 109: 3365-3370, 2012.
  • Ishikawa, K., Nara, A., Matsumoto, K.*, Hanafusa, H. EGFR-dependent phosphorylation of Leucine-rich repeat kinase LRRK1 is important for proper endosomal trafficking of EGFR. Mol. Biol. Cell 23: 1294-1306, 2012.